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categories: ["longevity", "foundations"]categories: ["longevity", "foundations"]tags: ["aging", "forecasting", "life extension", "demography", "biogerontology", "mortality"]tags: ["aging", "forecasting", "life extension", "demography", "biogerontology", "mortality"]summary: "The hypothetical threshold at which medicine adds remaining life expectancy faster than time subtracts it, making death by aging recede indefinitely."summary: "The hypothetical threshold at which medicine adds remaining life expectancy faster than time subtracts it, making death by aging recede indefinitely."updated: "2026-07-27"updated: "2026-08-23"humanEvidence: "No intervention has been shown to slow human aging on a validated endpoint; gains in remaining life expectancy at older ages have run at roughly a year per decade, an order of magnitude short of the threshold."humanEvidence: "No intervention has been shown to slow human aging on a validated endpoint; gains in remaining life expectancy at older ages have run at roughly a year per decade, an order of magnitude short of the threshold."issues: ["The keyfacts figure for life expectancy lost per year at 65 carries no source."]issues: ["The keyfacts figure for life expectancy lost per year at 65 carries no source."]------lines 80–85 → 80–8966 unchanged lines not shown
## What the record shows## What the record shows The empirical position as of 2026 is unfavorable to near-term escape velocity.The empirical position as of 2026 is unfavorable to near-term escape velocity. ```figure{"key": "life-expectancy-owid", "caption": "The record the concept extrapolates from. Nearly all of this rise came from preventing early deaths, not slowing aging — gains at birth say little about the remaining-life-expectancy race LEV actually requires."}``` No intervention has been shown to extend maximum human lifespan, and none has been shown to slow human aging on a validated endpoint. The leading geroprotector candidates — [[rapamycin]], [[metformin]], [[senolytics]], NAD+ precursors — have strong rodent data and either thin, null, or absent human data on aging outcomes. The best-evidenced life-extending intervention available to an individual is still [[exercise-and-aging|physical activity]], which does not obviously compound.No intervention has been shown to extend maximum human lifespan, and none has been shown to slow human aging on a validated endpoint. The leading geroprotector candidates — [[rapamycin]], [[metformin]], [[senolytics]], NAD+ precursors — have strong rodent data and either thin, null, or absent human data on aging outcomes. The best-evidenced life-extending intervention available to an individual is still [[exercise-and-aging|physical activity]], which does not obviously compound. removed, struck through added, underlinedLine numbers count the serialised markdown of each revision, frontmatter included.
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